Category BI L06 Investigating Drug Resistance in MCF7 WT and MCF60 Breast

Cancer Cell Lines

Abstract Breast cancer is the second most common cause of death for women

worldwide. Every year in the United States, 200,000 women and men are

diagnosed with breast cancer. Arsenic trioxide (As2O3) is currently used

to treat many types of cancer such as acute promyelocytic leukemia, and

the probable curative application of As2O3 to breast cancer is being

researched. Currently, cancer cells’ drug resistance to arsenic poses as a

major obstacle in the treatment of cancer.

The purpose of my project was to identify the arsenic transport systems

present in both wild type and arsenic resistant breast cancer cell lines

(MCF7 and MCF60 cell lines respectively). This project also investigated

the biochemical properties of the MCF60 arsenic resistant cell line in

comparison to the wildtype MCF7 line and determined the rate of uptake

and efflux of arsenic in both cell lines. MTT assays, transport experiments,

RNA isolation, and RT-PCR were all methods that were used to investigate

drug resistance and uptake. MTT assays were conducted to determine

cell counts after other cancer treating drugs such as doxorubicin and

etoposide were added to the cells for several days. EC50 values were

used as a comparison between the two cell lines and greater viable cell

counts were found from the MCF60 resistant cell line in comparison to the

MCF7 wild type cell line when treated with arsenic, antimony, and

pretreated with a BSO inhibitor. Transport experiments on the MCF7 cell

line were conducted to determine the cell’s rate of uptake at certain time

intervals. RNA was also isolated and reverse transcribed into cDNA, so

that the genes and proteins involved in arsenic resistance/efflux and

uptake were determined. The aquaporin protein AQP9 was found to

possibly play a role in arsenic uptake and efflux.

Overall, the results of this study suggest that the presence of AQP9

plays a role in the cell’s resistance to arsenic, as well as other drugs such

as antimony. It also concludes that the protein MRP is not involved in the

extrusion system of the MCF60 cell. This knowledge can help to solve the

problem of drug resistance and help continue the treatment of breast

cancer cells using heavy metals such as AsIII.

Bibliography Arsenic Trioxide in the mechanism of drug resistance reversal in MCF-

7/ADM cell line of human breast cancer. Wang X, Kong L, Zhao J, Yang P.

PubMed: 152836393.Differential effects of quercetin and silymarin on

arsenite-induced cytotoxicity in two human breast cancer cell lines. Soria

EA, Eynard AR, Quiroga PL, Bongiovanni GA. PMID: 17931660
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